Why primacor is deleterious in IHSS,does mustard operation work?
myomectomy of the septum
Asked by Ione Malahan 1 year ago.
The mustard won't work. You need either a septal ablation with alcohol (done in the cath lab) or an open myomectomy by a surgeon. Positive inotropes in general will increase the outflow obstruction in IHSS. Answered by Laverne Speelman 1 year ago.
I love mustard. I just love it. Answered by Jesica Bossenbroek 1 year ago.
What is the dosage of Milrinone - PRIMACOR in Cardiac Surgery?
Asked by Rodolfo Spinar 1 year ago.
Phosphodiesterase inhibitor with positive inotropic and vasodilator activity. Venodilator: 0 Arterial dilator: ++ Inotropic effect: +++ Calculation of drip rate: 50 mg/250ml (ml/hr) = wt (kg) x 0.3 x mcg/kg/min. Dosing (Adult): CHF: initial loading dose, 50 mcg/kg IV over 10min, then 0.375 to 0.75 mcg/kg/min IV (Usual: 0.5 mcg/kg/min). Cardiac surgery: 15min before separation from cardiopulmonary bypass, 50 mcg/kg IV over 20 minutes followed by a continuous infusion of 0.5 mcg/kg/min IV for a minimum of 4hr. Dose can vary according to patient status. Answered by Chau Dismuke 1 year ago.
How to maintain cardiac output?
I am a nursing student and I am doing a care plan about my patient. He was bedridden on a ventilator in a comatose state. What can I do to help him maintain optimal cardiac output?
Asked by Patience Weader 1 year ago.
To answer your question more information is needed. Is this a long term ventilator pt or pt in ICU/CCU? What other information did you get from your chart audit. What is his cardiac background? HTN, previous heart failure or heart attack? Is he currently dehydrated - what are his electrolytes. Research frank starling law, preload and afterload for heart, heart rate and stroke volume. As far as nursing diagnosis go keeping pt hydrated and heartbeat WNL and administering meds per MD either beta blockers - lopressor, ACE inhibitors- captopril, calcium channel blockers or in the CCU setting IV primacor or other IV meds dobutamine. A increased temperature increases heart rate which then changes cardiac output. A agitated pt with a increased heart rate can not fill his heart with enough blood in-between shortened heartbeat time to provide enough to pump forward and decreases cardiac output. Pain can increase heart rate and cause the same. 1) maintain fluid volume, monitor I & O, monitor electrolytes, blood sugar (increased BS depletes fluid in body) 2) maintain normal temperature 3) maintain pt comfort - just because he is comatose doesn't mean he cant feel pain. Pain is a basic neuro function. Does he still grimaces to painful stimuli? How comatose is he on glascow scale? 4) administer meds per M.D. 5) report abnormal VS to MD 6) report adverse reactions of meds to MD. 7) maintain proper ventilator settings / monitor arterial blod gases and report abnormals to MD. 8) provide proper "pulmonary toilet" means keeping pt suctioned and airway clear. 9) provide passive range of motion/ keep pt turned to prevent bedsores to prevent infection( increases temperature) GOOD LUCK! Answered by Clarinda Wheeley 1 year ago.
Im 44, my avg heart rate in the middle of my excercise routine is 173-176, When I push harder I get it up to around 185-187 and keep that intensity for 5-8 minutes, going back down to the 170's Love the feel of euphoria I get once I push past the 185 mark, Answered by Chieko Lefleur 1 year ago.
Are cardiac glycosides the same as cardiotonics?
I did research on both of them and to me, they have the same mechanism of action and do the exact same thing so I thought they may be the same "group" of drugs but with a different name. But if they're not.... what is another name for cardiotonic drugs?
Asked by Lilia Strate 1 year ago.
Inotropics and cardiotonics are medications that increase the strength of the muscle contractions that pump blood from the heart. All cardiac glycosides are cardiotonic drugs. But glycosides are not the only cardiotonic drugs. Cardiotonics are a group with initially mentioned characteristics of which cardiac glycosides are a sub-group. Until recently, the inotropic drug digitalis was the main cardiotonic. Now there are others such as: # Amrinone (Inocor®): A positive inotropic cardiotonic (CARDIOTONIC AGENTS) with vasodilator properties, phosphodiesterase inhibitory activity, and the ability to stimulate calcium ion influx into the cardiac cell. Used in CHF. # Dobutamine (Dobutrex®): Dobutamine is a sympathomimetic drug used in the treatment of heart failure and cardiogenic shock. Its primary mechanism is direct stimulation of β1 receptors of the sympathetic nervous system. # Milrinone (Primacor®): It has positive inotropic, vasodilating and minimal chronotropic effects. It is used in the management of heart failure only when conventional treatment with vasodilators and diuretics has proven insufficient. Answered by Crystal Henderson 1 year ago.
What Do Cardiac Glycosides Do Answered by Rosalinda Avellano 1 year ago.
Hello, As we move into the busy holiday season, I hope you’ll take a few minutes to read our latest newsletter. In this edition, I’ve taken a hard look at heart disease and heart attacks, with a close examination of the theories that form the basis of much of today’s treatment. I’m particularly pleased to share with you a report from a dynamic study group in Brazil, Infarctcombat.org. If you have any interest in this topic, I think you’ll find the information of great value. As an addition to the newsletter, I’ve included a book review this round, something I’ve been wanting to do for months. I hope you’ll find this useful as well. No holiday season is complete without a cheery and nutritious recipe, this time a very simple but delicious Cranberry sauce. Enjoy! Warmest wishes to you and your family for the holiday and coming year. Tom Cowan Answered by Earline Galarza 1 year ago.
yes. They are both positive inotrope drugs which means they both increase the strength of contraction of the heart. You can probably group them as Positive inotropes... I may be wrong about this because its midnight and I'm tired but I believe Cardiotonic drugs (they arent used as far as I know) but they can be grouped with beta 1 adrenergic agonists . sorry probably wasnt any help Answered by Joni Pfohl 1 year ago.
What does noncardiac pulmonary edema do to your body?
Asked by Merrie Dudack 1 year ago.
Non-heart-related pulmonary edema is caused by lung problems like pneumonia, an excess of intravenous fluids, some types of kidney disease, bad burns, liver disease, nutritional problems, and Hodgkin's disease. Non-heart-related pulmonary edema can also be caused by other conditions where the lungs do not drain properly, and conditions where the respiratory veins are blocked. Early symptoms of pulmonary edema include: shortness of breath upon exertion sudden respiratory distress after sleep difficulty breathing, except when sitting upright coughing In cases of severe pulmonary edema, these symptoms will worsen to: labored and rapid breathing frothy, bloody fluid containing pus coughed from the lungs (sputum) a fast pulse and possibly serious disturbances in the heart's rhythm (atrial fibrillation, for example) cold, clammy, sweaty, and bluish skin a drop in blood pressure resulting in a thready pulse Diagnosis A doctor can usually diagnose pulmonary edema based on the patient's symptoms and a physical exam. Patients with pulmonary edema will have a rapid pulse, rapid breathing, abnormal breath and heart sounds, and enlarged neck veins. A chest x ray is often used to confirm the diagnosis. Arterial blood gas testing may be done. Sometimes pulmonary artery catheterization is performed to confirm that the patient has pulmonary edema and not a disease with similar symptoms (called adult respiratory distress syndrome or "noncardiogenic pulmonary edema"). Treatment Pulmonary edema requires immediate emergency treatment. Treatment includes: placing the patient in a sitting position, oxygen, assisted or mechanical ventilation (in some cases), and drug therapy. The goal of treatment is to reduce the amount of fluid in the lungs, improve gas exchange and heart function, and, where possible, to correct the underlying disease. To help the patient breathe better, he/she is placed in a sitting position. High concentrations of oxygen are administered. In cases where respiratory distress is severe, a mechanical ventilator and a tube down the throat (tracheal intubation) will be used to improve the delivery of oxygen. Non-invasive pressure support ventilation is a new treatment for pulmonary edema in which the patient breathes against a continuous flow of positive airway pressure, delivered through a face or nasal mask. Non-invasive pressure support ventilation decreases the effort required to breath, enhances oxygen and carbon dioxide exchange, and increases cardiac output. Drug therapy could include morphine, nitroglycerin, diuretics, angiotensin-converting enzyme (ACE) inhibitors, and vasodilators. Vasopressors are used for cardiogenic shock. Morphine is very effective in reducing the patient's anxiety, easing breathing, and improving blood flow. Nitroglycerin reduces pulmonary blood flow and decreases the volume of fluid entering the overloaded blood vessels. Diuretics, like furosemide (Lasix), promote the elimination of fluids through urination, helping to reduce pressure and fluids in the blood vessels. ACE inhibitors reduce the pressure against which the left ventricle must expel blood. In patients who have severe hypertension, a vasodilator such as nitroprusside sodium (Nipride) may be used. For cardiogenic shock, an adrenergic agent (like dopamine hydrochloride [Intropin], dobutamine hydrochloride [Dobutrex], or epinephrine) or a bipyridine (like amrinone lactate [Inocor] or milrinone lactate [Primacor]) are given. Prognosis Most patients with pulmonary edema who seek immediate treatment can be treated quickly and effectively. ______________________ Answered by Maxie Arguellez 1 year ago.
Pulmonary edema is literally the deposition of fluid into the alveoli which are the end points of the smallest airways. This fluid prevents the exchange of oxygen and carbon dioxide. Pulmonary edema is a very serious condition which may lead to death in a sense from 'drowning'. Although I do not watch must television I will admit that I enjoy the cerebral Las Vegas CSI which had an episode of death due to altitude related pulmonary edema. Indeed rapid ascent to as little as 10 000 feet (think of the rim of the Grand Canyon) may induce pulmonary edema and I have treated an individual returning from that very circumstance. Answered by Lory Sagen 1 year ago.
This Site Might Help You. RE: What does noncardiac pulmonary edema do to your body? Answered by Malka Brazzle 1 year ago.
It makes your body retain (keep inside) fluids. It can cause shortness of breath and other breathing problems, that make you tired and weak feeling. You should see a pulmonary (Lung) doctor for that. Answered by Damion Dorshimer 1 year ago.
www.healthatoz.com/healthatoz/Atoz/ency/... Above is some great info on the subject, it causes extreme swelling. Answered by Aldo Barona 1 year ago.