Application Information

This drug has been submitted to the FDA under the reference 012265/003.

Names and composition

"NAQUIVAL" is the commercial name of a drug composed of RESERPINE and TRICHLORMETHIAZIDE.

Forms

ApplId/ProductId Drug name Active ingredient Form Strenght
012265/003 NAQUIVAL RESERPINE; TRICHLORMETHIAZIDE TABLET/ORAL 0.1MG and 4MG

Similar Active Ingredient

ApplId/ProductId Drug name Active ingredient Form Strenght
012265/003 NAQUIVAL RESERPINE; TRICHLORMETHIAZIDE TABLET/ORAL 0.1MG and 4MG
012972/001 METATENSIN #2 RESERPINE; TRICHLORMETHIAZIDE TABLET/ORAL 0.1MG and 2MG
012972/002 METATENSIN #4 RESERPINE; TRICHLORMETHIAZIDE TABLET/ORAL 0.1MG and 4MG
085248/001 TRICHLORMETHIAZIDE W/ RESERPINE RESERPINE; TRICHLORMETHIAZIDE TABLET/ORAL 0.1MG and 4MG

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Answered questions

What can make neurons release and degrade neurotransmitters INSIDE the axon terminal?
Is there any toxin, poison, chemical, or man-made drug capable of this? I thought I remembered hearing something about a toxin capable of this in a neurosci class, but I can't find anything on the internet right now and I've been searching for hours without luck. I also tried searching under... Asked by Judi Rocray 1 year ago.

Is there any toxin, poison, chemical, or man-made drug capable of this? I thought I remembered hearing something about a toxin capable of this in a neurosci class, but I can't find anything on the internet right now and I've been searching for hours without luck. I also tried searching under "Intracytoplasmic disintegration of synaptic vesicles" but google is not being kind to me today. Answered by Rosann Salvetti 1 year ago.

I.Drug Name: Naquival ( Reserpine) II.Effects: It works by inhibiting the ATP/Mg2+ pump that is in charge of moving norepinephrine into storage vesicles in the presynaptic neuron. These non-packaged neurotransmitters are then readily degraded by MAO (monoamine oxidase). Results in the reduction in catecholamines and serotonin from the central and peripheral axon terminals. Answered by Damien Mart 1 year ago.

I haven't heard anything as such, though enzymatic deactivation comes to mind as it causes the destruction of a neurotransmitter by an enzyme after its release. Some drugs acts as an agonist in that the chemical release of the drug stops the vesicle membrane of the presynaptic neuron to reuptake in that it sends the neurotransmitter back to the terminal buttons thus stoping the postsynaptic potential. Drugs such as Chlorpromazine used to block the D2 dopomine receptor. Answered by Amina Zahnow 1 year ago.


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