My husband is on hydrea for polycythemia. That is a chemo medicine. Can he pass the meds to me during sex?
Asked by Nilda Lecher 1 year ago.
Many oncologists recommend that condoms be used to prevent transfer of chemo agents during sex. It would be best to ask his onc if this particular chemo is a concern for this to occur. Answered by Quiana Etheridge 1 year ago.
Regardless, if he is on chemo you need to be using condoms anyway. His immune system is down, and that will make him more susceptable to infections. Even if you guys are monogamous/std free, he could get a bacterial infection. The best source however, is to ask his doctor. He should also ask his doctor is anal penetration is appropriate now. Anal penetration can cause tears in the wall of the rectum which leaves opportunity for bacterial infection because his immune system is low and bleeding if the platelet count is low. Answered by Roni Ardizzone 1 year ago.
That is an excellent question, Have your husband call the doctor and ask. My dad had polyythemia and was on that med for many years and lived a long time with the disease..Good luck Answered by Tony Clizbe 1 year ago.
My husband's body is not responding well to Hydrea for Polycythemia? What else can I do to help him?
Asked by Kellee Sarra 1 year ago.
-In yahoo answers I have read very good discussions about cancer . You too might have read a lot of information about cancer here in yahoo answers. Hence ,If you go on writing answers to the questions asked by many questioners about cancer here in yahoo answers ,then ,I can understand how much knowledge you have already gained about cancer .If there if mistake in any of your way of thinking about cancer ,I can write my opinion about cancer to you. If I feel that you have not given sufficient importance to any important point about cancer ,I can write about that to you You should try to understand each and every sentence written about cancer ,here in yahoo answers. If you do not understand any difficult English word you should refer a dictionary to find out the meaning Now I have to know how many information you already know about cancer. Then I can understand ,what are the information you do not know about cancer .I should know about to have also If you have not read those pages already ,then I have to write to you those information. If you have already read those pages then if I write again those information ,it will be boring., uninteresting topic to you .Therefore ,I will have to write to you about more advanced topics, What I have to write to you depends on how many hours you have already read about cancer. It will be useful if I know the type of cancer the patient is suffering from ,duration elapsed since commencement and the name of the medicines the patient is taking already . -You know that you can write e mails to me by clicking the word e mail which is visible in my profile available by clicking here my name/picture Answered by Apolonia Batcheller 1 year ago.
you need a real energy consumption, the rest of the health or well to meet the needs that set wife. Answered by Susana Wenrich 1 year ago.
What are treatment options for sickle cell disease? How do the treatments affect the cells and the individual?
Asked by Emory Dewoody 1 year ago.
Hydroxyurea (Hydrea) Folic Acid Hydration These are the 3 coner stones of keeping the patient well and preventing crisis. Answered by Marguerita Hausladen 1 year ago.
I was dx with essential thrombocythemia 14 years ago. My platelets have taken a dive! What could this mean?
I've tried to find what it means when my platelets dive from around 600 or 700 to a low of 141 over just a few months. Has my ET transformed into something worse. I have been taking Hydrea for 14 years. Any ideas?
Asked by Jeanene Schroll 1 year ago.
Talk to a specialist as soon as you can! You can never be too safe. ET can rarely evolve into leukemia, a sign of which is a drastic drop in platelets. So get it checked out ASAP! Answered by Brett Augustin 1 year ago.
If you are familiar with leukemia treatments I would appreciate your insights.?
Would it be save to say that with leukemia that one is either getting better or getting worst? Is there any such thing as holding your own? A 49 yr old male with CML with a fairly rapid onset of symptoms
Asked by Rivka Frese 1 year ago.
If you have started Gleevec you should see results within a few (3-4) weeks. If you had a very aggressive presentation, your docs may be using other things to get it under control, such as pheresis or hydrea. Even though your symptoms increased rapidly, you have likely had the disease for several months already, at least. It can just take this long for the disease to grow enough for the symptoms to show. Even if Gleevec does not work for you, some of the second generation TK inhibitors can work just as well. I have several patients who have been on dasatinib for a long tome and are doing very well. Get a consultation with a transplant specialist as well, to learn what your options are for later on. But I have patients who have been on Gleevec for over 5 years and are still in perfect control and may never need a transplant. Best of luck to you. Answered by Steve Meltz 1 year ago.
Unfortunately I did not begin to see results for 2 months. Of course I had a lot of obstacles to overcome but that is how long it took me and I would say that that was average. Answered by Kristina Linssen 1 year ago.
Unfortunately, everyone reacts differently to the chemotherapy and it would be impossible to say what type of results you will have and in what time frame. Ask your oncologist on your next visit. Good luck. Answered by Milissa Dupaski 1 year ago.
What is the major of chemotherapy agents in anti-cancer drugs ?
Asked by Curtis Morath 1 year ago.
There are many different chemotherapy agents. Different drugs work for different cancers, and they are frequently used in combination. You need to be more specific. Here is a list of chemo drugs: 13-cis-Retinoic Acid 2-CdA 2-Chlorodeoxyadenosine 5-Fluorouracil 5-FU 6-Mercaptopurine 6-MP 6-TG 6-Thioguanine Abraxane Accutane ® Actinomycin-D Adriamycin ® Adrucil ® Agrylin ® Ala-Cort ® Aldesleukin Alemtuzumab ALIMTA Alitretinoin Alkaban-AQ ® Alkeran ® All-transretinoic acid Alpha interferon Altretamine Amethopterin Amifostine Aminoglutethimide Anagrelide Anandron ® Anastrozole Arabinosylcytosine Ara-C Aranesp ® Aredia ® Arimidex ® Aromasin ® Arranon ® Arsenic trioxide Asparaginase ATRA Avastin ® Azacitidine BCG BCNU Bevacizumab Bexarotene BEXXAR ® Bicalutamide BiCNU Blenoxane ® Bleomycin Bortezomib Busulfan Busulfex ® C225 Calcium Leucovorin Campath ® Camptosar ® Camptothecin-11 Capecitabine Carac ™ Carboplatin Carmustine Carmustine wafer Casodex ® CC-5013 CCNU CDDP CeeNU Cerubidine ® Cetuximab Chlorambucil Cisplatin Citrovorum Factor Cladribine Cortisone Cosmegen ® CPT-11 Cyclophosphamide Cytadren ® Cytarabine Cytarabine liposomal Cytosar-U ® Cytoxan ® Dacarbazine Dacogen Dactinomycin Darbepoetin alfa Daunomycin Daunorubicin Daunorubicin hydrochloride Daunorubicin liposomal DaunoXome ® Decadron Decitabine Delta-Cortef ® Deltasone ® Denileukin diftitox DepoCyt ™ Dexamethasone Dexamethasone acetate Dexamethasone Sodium Phosphate Dexasone Dexrazoxane DHAD DIC Diodex Docetaxel Doxil ® Doxorubicin Doxorubicin liposomal Droxia ™ DTIC DTIC-Dome ® Duralone ® Efudex ® Eligard ™ Ellence ™ Eloxatin ™ Elspar ® Emcyt ® Epirubicin Epoetin alfa Erbitux ™ Erlotinib Erwinia L-asparaginase Estramustine Ethyol Etopophos ® Etoposide Etoposide Phosphate Eulexin ® Evista ® Exemestane Fareston ® Faslodex ® Femara ® Filgrastim Floxuridine Fludara ® Fludarabine Fluoroplex ® Fluorouracil Fluorouracil (cream) Fluoxymesterone Flutamide Folinic Acid FUDR ® Fulvestrant G-CSF Gefitinib Gemcitabine Gemtuzumab ozogamicin Gemzar ® GleevecTM Gliadel wafer (t) GM-CSF Goserelin granulocyte - colony stimulating factor (t) Granulocyte macrophage colony stimulating factor (o) Halotestin (t) Herceptin (t) Hexadrol (t) Hexalen (t) Hexamethylmelamine (t) HMM (t) Hycamtin (t) Hydrea (t) Hydrocort Acetate (t) Hydrocortisone Hydrocortisone sodium phosphate Hydrocortisone sodium succinate Hydrocortone phosphate (t) Hydroxyurea Ibritumomab Ibritumomab Tiuxetan Idamycin ® Idarubicin Ifex ® IFN-alpha Ifosfamide IL-11 IL-2 Imatinib mesylate Imidazole Carboxamide Interferon alfa Interferon Alfa-2b (PEG conjugate) (o) Interleukin - 2 (t) Interleukin-11 (o) Intron A® (interferon alfa-2b) Iressa ® Irinotecan Isotretinoin Kidrolase (t) Lanacort (t) L-asparaginase (t) LCR (o) Lenalidomide Letrozole Leucovorin Leukeran (t) Leukine (t) Leuprolide Leurocristine (o) Leustatin (t) Liposomal Ara-C (t) Liquid Pred (t) Lomustine L-PAM (o) L-Sarcolysin (o) Lupron (t) Lupron Depot ® Matulane (t) Maxidex (t) Mechlorethamine Mechlorethamine Hydrochloride Medralone (t) Medrol ® Megace (t) Megestrol Megestrol Acetate (o) Melphalan Mercaptopurine Mesna Mesnex (t) Methotrexate Methotrexate Sodium (o) Methylprednisolone Meticorten (t) Mitomycin Mitomycin-C (o) Mitoxantrone M-Prednisol (t) MTC (o) MTX (o) Mustargen (t) Mustine Mutamycin (t) Myleran (t) Mylocel (t) Mylotarg (t) Navelbine ® Nelarabine Neosar (t) Neulasta (t) Neumega (t) Neupogen ® Nexavar ® Nilandron (t) Nilutamide Nipent ® Nitrogen Mustard (o) Novaldex (t) Novantrone (t) Octreotide Octreotide acetate (o) Oncospar (t) Oncovin (t) Ontak (t) Onxal (t) Oprevelkin Orapred (t) Orasone (t) Oxaliplatin Paclitaxel Paclitaxel Protein-bound Pamidronate Panretin (t) Paraplatin (t) Pediapred (t) PEG Interferon Pegaspargase Pegfilgrastim PEG-INTRON (t) PEG-L-asparaginase PEMETREXED Pentostatin Phenylalanine Mustard (o) Platinol (t) Platinol-AQ (t) Prednisolone Prednisone Prelone (t) Procarbazine PROCRIT ® Proleukin (t) Prolifeprospan 20 with Carmustine implant (t) Purinethol ® Raloxifene Revlimid ® Rheumatrex (t) Rituxan (t) Rituximab Roferon-A® (interferon alfa-2a) Rubex (t) Rubidomycin hydrochloride (t) Sandostatin ® Sandostatin LAR (t) Sargramostim Solu-Cortef (t) Solu-Medrol (t) Sorafenib STI-571 Streptozocin SU11248 Sunitinib Sutent ® Tamoxifen Tarceva ® Targretin (t) Taxol ® Taxotere ® Temodar ® Temozolomide Teniposide TESPA (o) Thalidomide Thalomid ® TheraCys (t) Thioguanine Thioguanine Tabloid ® Thiophosphoamide (o) Thioplex (t) Thiotepa TICE ® Toposar (t) Topotecan Toremifene Tositumomab Trastuzumab Tretinoin Trexall (t) Trisenox (t) TSPA (o) VCR (o) Velban (t) Velcade ® VePesid (t) Vesanoid (t) Viadur (t) Vidaza (t) Vinblastine Vinblastine Sulfate (o) Vincasar Pfs (t) Vincristine Vinorelbine Vinorelbine tartrate (o) VLB (o) VM-26 (o) VP-16 (t) Vumon (t) Xeloda ® Zanosar (t) Zevalin TM Zinecard (t) Zoladex ® Zoledronic acid Zometa ® See? There's a lot of them. Answered by Hipolito Crow 1 year ago.
antineoplastics, monoclonal antibodies, Answered by Bobbie Pevsner 1 year ago.
Please see the webpages for more details on Chemotherapy. Answered by Charlie Gerock 1 year ago.
What does it mean if someone has a higher than average level of platelets in their blood?
What are the causes and what does it mean. Someone has mentioned Leukemia to me and now I'm panicking a little. The doctor has said to go back in 6 weeks for another blood test.
Asked by Kasandra Manocchia 1 year ago.
Thrombocythemia (high platelet count) Thrombocythemia refers to a high platelet count. Platelets (thrombocytes) play an important role in blood clotting. The term "essential thrombocythemia" means that the cause of a high platelet count can't be determined. A normal platelet count ranges from 150,000 to 450,000 platelets per microliter of blood. In very large numbers — exceeding 600,000 platelets per microliter — platelets may cause clotting in blood vessels and abnormal bleeding. A doctor may diagnose essential thrombocythemia by blood tests that show a sustained increase in platelets not due to another condition, such as polycythemia vera or chronic myelogenous leukemia. In some cases, thrombocythemia is due to a specific genetic mutation. Some people with thrombocythemia have no signs or symptoms. Others may experience bleeding from the nose or gums, blood clots, heart attack or stroke. When needed, treatment may include: * Low-dose aspirin to reduce clotting risk * Medications such as interferon or hydroxyurea (Hydrea, Droxia) to lower platelet counts in those individuals who are older than age 60, are at high risk of heart disease or have a history of abnormal bleeding or blood clots Essential thrombocythemia is a stem cell disease; "essential" means that the cause is unknown. Blood cells, including red blood cells (which carry oxygen from the lungs to the rest of the body), white blood cells (which fight infections) and platelets (which form blood clots) are produced from stem cells in the bone marrow. The stem cells grow and divide into intermediate cells and finally into mature cells in the blood. A problem with the stem cell can lead to problems with all other cells. ET is a stem cell disease that leads to an overproduction of platelets (thrombocytes) without other secondary causes, sometimes with an accompanying increase in white blood cells. Blood platelets help prevent and control bleeding resulting from injury to blood vessels. The term thrombocythemia refers to an unusually high platelet count. A normal count ranges from 150,000 to 450,000 platelets per microliter (mL) of blood. When the platelet count exceeds 600,000 platelets per mL, doctors call this essential thrombocythemia. Most young people with essential thrombocythemia have no signs or symptoms. ET is classified as a chronic myeloproliferative disorder (CMPD). The CMPDs also include polycythemia vera, agnogenic myeloid metaplasia with myelofibrosis, and chronic myelogenous leukemia. Answered by Kattie Rafala 1 year ago.
For decades the white blood count and differential were done manually, with the tech staining a blood smear and physically counting the cells. The first automatic counters only counted the total WBC count and the differential was still performed manually. Platelets were estimated as low, normal or increased. If you wanted more info, you ordered a special platelet count. Somewhere around 1980 or thereabouts automated counters came along that could give you an accurate total WBC plus a differential, plus platelets. Surprisingly it took a fairly long time for some of us to realize that platelets were "acute phase reactants" that rose and fell with fever and stress. So while yes, there are disorders associated with increased platelets (see a previous response), it can also be just a reaction to stress. Answered by Mariel Chaton 1 year ago.
It's unlikely to be leukemia as it tends to lower the platelet count. Answered by Annis Babbel 1 year ago.
Sickle Cell Anemia................?
Does anybody have any information on sickle cell anemia like treatments, how it affects the person, support groups. Its for this big project im doin for biology worth about 50% of our term grade. Please help and also list resources.
Asked by Dara Aubrey 1 year ago.
Treatments and drugs Bone marrow transplant offers the only potential cure for sickle cell anemia. But very few people have a suitable donor for transplant. As a result, treatment for sickle cell anemia is usually aimed at avoiding crises, relieving symptoms and preventing complications. If you have sickle cell anemia, you'll need to make regular visits to your doctor to check your red blood cell count and monitor your health. You may also require treatment from specialists at a hospital or sickle cell anemia clinic. Treatments may include medications to reduce pain and prevent complications, blood transfusions and supplemental oxygen, as well as bone marrow transplant. Medications Medications used to treat sickle cell anemia include: Antibiotics. Children with sickle cell anemia usually begin taking the antibiotic penicillin when they're about 2 months of age and continue until they're 5 years old. Doing so helps prevent infections, such as pneumonia, which can be life-threatening to an infant or child with sickle cell anemia. Antibiotics may also help adults with sickle cell anemia fight certain infections. Pain-relieving medications. To relieve pain during a sickle crisis, your doctor may advise over-the-counter pain relievers and application of heat to the affected area. You may also need stronger prescription painkillers. Hydroxyurea (Droxia, Hydrea). This prescription drug, normally used to treat cancer, may be helpful for adults with severe disease. When taken daily, it reduces the frequency of painful crises and may reduce the need for blood transfusions. It seems to work by stimulating production of fetal hemoglobin — a type of hemoglobin found in newborns that helps prevent the formation of sickle cells. There is some concern about the possibility that long-term use of this drug may cause tumors or leukemia in certain people. Your doctor can help you determine if this drug may be beneficial for you. Blood transfusions In a red blood cell transfusion, red blood cells are removed from a supply of donated blood. These donated cells are then given intravenously to a person with sickle cell anemia. Blood transfusions increase the number of normal red blood cells in circulation, helping to relieve anemia. In children with sickle cell anemia at high risk of stroke, regular blood transfusions can decrease their risk of stroke. Blood transfusions carry some risk. Blood contains iron. Regular blood transfusions cause an excess amount of iron to build up in your body. Because excess iron can damage your heart, liver and other organs, people who undergo regular transfusions must often receive treatment to reduce iron levels. In 2005, the Food and Drug Administration approved deferasirox (Exjade), the first oral medication that can reduce excess iron levels, for use in people older than the age of 2. Supplemental oxygen Breathing supplemental oxygen through a breathing mask adds oxygen to your blood and helps you breathe easier. It may be helpful if you have acute chest syndrome or a sickle cell crisis. Bone marrow transplant This procedure allows people with sickle cell anemia to replace their bone marrow — and its sickle-shaped red blood cells — with healthy bone marrow from a donor who doesn't have the disease. It can be a cure, but the procedure is risky, and it's difficult to find suitable donors. Researchers are still studying bone marrow transplants for people with sickle cell anemia. Currently, the procedure is recommended only for people who have significant symptoms and problems from sickle cell anemia. In a bone marrow transplant, your bone marrow is first destroyed using chemotherapy or radiation. A matched donor's healthy bone marrow is removed using a minor surgical technique. You then receive a transfusion of that healthy marrow, and the healthy marrow may start producing normal blood cells. Sometimes, however, the transplant doesn't work or the recipient's body rejects the new marrow. The procedure requires a lengthy hospital stay. After the transplant, you'll need drugs to help prevent rejection of the donated marrow. Treating complications Doctors treat most complications of sickle cell anemia as they occur. Treatment may include antibiotics, blood transfusions, pain-relieving medicines, other medications and possibly surgery, such as to correct vision problems or to remove a damaged spleen. Experimental treatments Scientists continue to gain new insights into the symptoms and causes of sickle cell anemia. Some possible new treatments researchers are studying include: Gene therapy. Because sickle cell anemia is caused by a defective gene, researchers are exploring whether correcting this gene and inserting it into the bone marrow of people with sickle cell anemia will result in the production of normal hemoglobin. Scientists are also exploring the possibility of turning off the defective gene while reactivating another gene responsible for the Answered by Beverlee Drummey 1 year ago.